Doctors puzzled by baby born with Zika virus
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Zika-infected couples should postpone pregnancy: U.S. health officials
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Yale study identifies cells in human placenta that can carry Zika virus
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Zika study says virus passes placenta to foetus
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FRIDAY, May 27, 2016 (HealthDay News) New research seems to shed light on how the Zika virus infects, but doesn’t kill, placenta cells. The mosquito-borne virus can cause severe birth defects in babies whose mothers are exposed to Zika during pregnancy, but scientists don’t know exactly how that happens. The researchers found that Zika can replicate in immune cells from the placenta without killing them. They said this may explain how the virus can pass through the placenta of a pregnant woman and.
Zika virus can infect and replicate in immune cells from the placenta, without killing them, scientists have discovered. The finding may explain how the virus can pass through the placenta of a pregnant woman, on its way to infect developing brain cells in her fetus. The results are published in.
Zika virus can infect and replicate in immune cells from the placenta, without killing them, researchers, including one of Indian-origin, have found. The finding may explain how the virus can pass through the placenta of a pregnant woman, on its way to infect developing brain cells in her foetus. New York: A team of researchers led by Indian-origin scientists have discovered that the deadly Zika virus can infect and replicate in immune cells from the placenta, without killing them. Two studies recently published in Cell showed, in experiments conducted on female mice, how Zika is able to cross the placenta, infect the fetus and cause microcephaly—insufficient development of.
Decreased oxygen levels in a placenta can impair fetal development and ultimately the health of a baby after birth. This artist’s image of a placenta shows how the Zika virus can affect a mother’s womb. The maternal blood vessels that feed into the placenta (A) become narrower and limit blood flow to the fetus.
Zika, on the other hand, is already active. It is released from an infected cell and then infects others, including placental cells that contact the fetal bloodstream. It can very quickly — and very early — reach fetal blood inside the developing placenta.
The two viruses are quite different from each other. An alternative hypothesis is that non-neutralizing, cross-reactive antibodies bind ZIKV and traffic across the placenta, through a neonatal Fc-receptor-mediated mechanism, to infect placental macrophages. Various cell types in the placenta are susceptible to infection with Zika virus. Specifically, observations of hydropic and hyperplastic chorionic villi, as well as the proliferation of Hofbauer cells, have been reported during infection with Zika virus [ 12 ].
List of related literature:
|from Pregnant in the Time of Ebola: Women and Their Children in the 2013-2015 West African Epidemic|
|from Chestnut’s Obstetric Anesthesia E-Book|
|from Robbins & Cotran Pathologic Basis of Disease E-Book|
|from Rheumatology Secrets E-Book|
|from Neonatology: A Practical Approach to Neonatal Diseases|
|from Male Infertility: Contemporary Clinical Approaches, Andrology, ART and Antioxidants|
|from Principles of Gender-specific Medicine|
|from Joints and Connective Tissues: General Practice: The Integrative Approach Series|
|from Diseases of Swine|
|from Maternal, Fetal, & Neonatal Physiology4: Maternal, Fetal, & Neonatal Physiology|